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Chronic obstructive pulmonary disease (COPD) is a major health problem. One of the major components of cigarette smoke (CS) is nicotine. Nicotine could induce inflammation and lung injury, whereas delphinidin is one of the main anthocyanin components in grapes. The present study was designed to investigate the protective effect of delphinidin in CS-induced pulmonary injury. C57BL/6 mice were randomly divided into four groups: (i) control mice, (ii) CS mice (CS group), (iii) CS mice treated with delphinidin (CS-Del group), and (iv) CS mice treated with a vehicle (CS-Veh group). The lung tissue was obtained to measure the concentrations of tumor necrosis factor-α (TNF-α), interleukin-1β (IL-1β) and IL-6, nitric oxide (NO), and superoxide dismutase (SOD) activity. The histological and pathological changes were observed. CS markedly increased the concentrations of TNF-α, IL-1β and IL-6, NO and SOD activity, and the severity of pathological changes, and decreased the pulmonary function. Delphinidin markedly attenuated CS-induced pulmonary inflammatory injury by reducing the levels of TNF-α, IL-1β and IL-6, and NO, and elevated SOD activity in the lungs. Delphinidin also ameliorated the pathological changes in lung tissues. Delphinidin attenuates CS-induced pulmonary inflammation and improves the pulmonary function. The protective effect of delphinidin on pulmonary inflammation may be associated with its antioxidant activity.Lindsay Lohan Dumps Her Producers and Could Return to Acting
She’s close to a deal that would allow her to return to movies after a brief but very public jail stint.
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